https://obstetricalpathology.com/letters-on-obstetrical-pathology-2/2024-12-11T18:19:27+00:00weekly0.6https://obstetricalpathology.com/2024/12/09/letters-on-obstetrical-pathology-loop-1-a-newsletter/2024-12-09T16:10:09+00:00monthlyhttps://obstetricalpathology.com/letters-on-obstetrical-pathology-loop-volume-1-number-1/2024-12-09T15:36:22+00:00weekly0.6https://obstetricalpathology.com/umbilical-cord-coiling-index/https://obstetricalpathology.com/wp-content/uploads/2024/01/image-1.jpegimage-1https://obstetricalpathology.com/wp-content/uploads/2024/01/image.jpegimagehttps://obstetricalpathology.com/wp-content/uploads/2024/01/image-1.pngimage-12024-01-15T02:44:30+00:00weekly0.6https://obstetricalpathology.com/cord-width/https://obstetricalpathology.com/wp-content/uploads/2024/01/whartons-jelly.pngwhartons-jellyhttps://obstetricalpathology.com/wp-content/uploads/2024/01/twin-cord-edema.jpegtwin-cord-edema2024-01-15T01:33:11+00:00weekly0.6https://obstetricalpathology.com/umbilical-cord-length-short-and-long-cords/https://obstetricalpathology.com/wp-content/uploads/2024/01/image.pngimagehttps://obstetricalpathology.com/wp-content/uploads/2024/01/11312.jpg11312https://obstetricalpathology.com/wp-content/uploads/2024/01/6154.jpg61542024-01-15T01:20:46+00:00weekly0.6https://obstetricalpathology.com/2023/03/12/a-study-protocol-of-stillbirth-in-need-of-a-pi/2023-03-12T13:44:44+00:00monthlyhttps://obstetricalpathology.com/sumt-stillbirth-umbilical-cord-mri-tipping-point-study/2023-03-12T13:37:54+00:00weekly0.6https://obstetricalpathology.com/2021/12/12/a-system-approach-to-stillbirth-perinatal-brain-injury-and-unexpected-deterioration-of-fetal-heart-rate-tracing/2021-12-12T18:55:21+00:00monthlyhttps://obstetricalpathology.com/revision-of-the-mri-stillbirth-study-protocol/2021-02-07T15:44:17+00:00weekly0.6https://obstetricalpathology.com/2021/02/07/autopsy-revision-to-the-mri-stillbirth-proposal/2021-02-07T15:41:24+00:00monthlyhttps://obstetricalpathology.com/2021/01/31/autopsy-brain-manual-and-next-step/2021-01-31T21:25:24+00:00monthlyhttps://obstetricalpathology.com/autopsy-manual-brain/2021-01-31T18:13:38+00:00weekly0.6https://obstetricalpathology.com/kidney-autopsy-manual/2021-01-24T14:38:12+00:00weekly0.6https://obstetricalpathology.com/2021/01/17/new-autopsy-manual-section-on-liver/2021-01-17T21:23:36+00:00monthlyhttps://obstetricalpathology.com/autopsy-manual-liver/2021-01-17T20:44:10+00:00weekly0.6https://obstetricalpathology.com/2021/01/05/new-autopsy-manual-page-thyroid/2021-01-05T15:56:05+00:00monthlyhttps://obstetricalpathology.com/autopsy-manual-thyroid/2021-01-05T15:51:26+00:00weekly0.6https://obstetricalpathology.com/2021/01/03/new-page-autopsy-manual-spleen/2021-01-03T13:36:55+00:00monthlyhttps://obstetricalpathology.com/autopsy-manual-spleen/2021-01-03T13:34:28+00:00weekly0.6https://obstetricalpathology.com/2020/12/27/new-post-autopsy-manual-lung/2020-12-27T14:44:45+00:00monthlyhttps://obstetricalpathology.com/autopsy-manual-lung/2020-12-27T14:31:13+00:00weekly0.6https://obstetricalpathology.com/2020/12/20/new-page-of-autopsy-manual-heart/2020-12-20T16:05:48+00:00monthlyhttps://obstetricalpathology.com/the-heart/2020-12-20T16:04:25+00:00weekly0.6https://obstetricalpathology.com/2020/12/09/stillbirth-mechanisms-and-causes/2020-12-09T14:58:45+00:00monthlyhttps://obstetricalpathology.com/2020/12/05/new-page-on-pancreas/2020-12-05T16:24:27+00:00monthlyhttps://obstetricalpathology.com/autopsy-manual-pancreas/2020-12-05T13:50:19+00:00weekly0.6https://obstetricalpathology.com/2020/11/29/new-page/2020-11-29T15:12:23+00:00monthlyhttps://obstetricalpathology.com/autopsy-manual-thymus/2020-11-29T15:06:31+00:00weekly0.6https://obstetricalpathology.com/2020/11/22/new-page-on-adrenals/2020-11-22T14:41:00+00:00monthlyhttps://obstetricalpathology.com/autopsy-manual-adrenals/2020-11-22T14:20:18+00:00weekly0.6https://obstetricalpathology.com/2020/11/15/chronic-histiocytic-intervillositis-study-proposal/2020-11-15T16:28:17+00:00monthlyhttps://obstetricalpathology.com/work-in-progress-digital-book/2020-11-15T16:20:08+00:00weekly0.6https://obstetricalpathology.com/2020/11/08/additions-to-intervillositis-digital-book-section/2020-11-08T15:03:44+00:00monthlyhttps://obstetricalpathology.com/2020/11/01/proposal-for-an-digital-book-on-placenta-pathology/2020-11-01T13:04:39+00:00monthlyhttps://obstetricalpathology.com/2020/10/18/new-page-added-an-invitation-to-study-stillbirth/2020-10-18T13:38:10+00:00monthlyhttps://obstetricalpathology.com/an-invitation-to-study-stillbirth/2020-10-18T13:33:35+00:00weekly0.6https://obstetricalpathology.com/umbilical-cord-accident-direct-compression/2020-07-03T14:58:54+00:00weekly0.6https://obstetricalpathology.com/2019/09/30/cesarean-section-association-with-autism-and-attention-deficit/https://obstetricalpathology.com/wp-content/uploads/2019/09/fig-6927.jpgfig 6927This photomicrograph from the region near CA1 of the hippocampus shows numerous karyorrhectic neurons (arrows) H&E 40x2019-09-30T20:44:46+00:00monthlyhttps://obstetricalpathology.com/chap-10a-villitis-of-unknown-etiology-vue-and-chronic-intervillositis/https://obstetricalpathology.com/wp-content/uploads/2019/09/11366.jpg11366This image is from the first pregnancy of a mother whose second infant died of the complications of Rh alloimmunization (Immune Hydrops). Is VUE part of the sensitization process? Fortunately, I did not have enough cases to explore this due to adequate postpartum therapy of RH- mothers.(H&E)https://obstetricalpathology.com/wp-content/uploads/2019/09/1101.jpg1101The central villus shows that the lymphocytes just beneath the syncytium are T-lymphocytes, and are associated with a breach in a the lower portion of the syncytium. (CD-3, 20x)https://obstetricalpathology.com/wp-content/uploads/2019/09/k4123.jpgk4123The is a placenta with coarsely granular villi especially in the middle slice that demonstrated extensive VUE.https://obstetricalpathology.com/wp-content/uploads/2019/08/ob-10a-fig-54-5862toxo.jpgOb 10a fig 54 5862toxoFig 54) This cluster of villi appears to be VUE, except perhaps for some very subtle differences such as few cells with larger, irregular nuclei, and some areas of coagulation necrosis in the villi. I don’t think prospectively that I would have recognized this for a villitis associated with toxoplasmosis infection of the infant, which it was. (H&E, 20x)https://obstetricalpathology.com/wp-content/uploads/2019/08/ob-10a-fig-53-7925.jpgOB 10a Fig 53 7925Fig 53) There is a partial thrombus in a main vessel of a small stem villus. The inflammation from VUE appears to be continuous with villi branching directly from it in the plane of section, and surrounded by other villi with VUE. (H&E 20x)https://obstetricalpathology.com/wp-content/uploads/2019/08/ob-10a-fig-52-2d.jpgOB 10a Fig 52 2DFIg 52) From the same case as Fig 51, C4d staining is present over the adjacent syncytiotrophoblast border. (C4d immunostain)https://obstetricalpathology.com/wp-content/uploads/2019/08/ob-10a-fig-51-mci2b.jpgOB 10a Fig 51 MCI2BFig 51) An area of massive chronic intervillositis (H&E)https://obstetricalpathology.com/wp-content/uploads/2019/08/ob-10a-fig-50-8396.jpgOB 10a Fig 50 8396Fig 50) At higher magnification in the same case as Fig 49, a cluster of intervillous monocytes can be seen.https://obstetricalpathology.com/wp-content/uploads/2019/08/ob-10a-fig-49-8397.jpgOB 10a Fig 49 8397Fig 49) The arrow points to a focal area of intervillous monocytes in this term, AGA uncomplicated pregancy (H&E,2x)https://obstetricalpathology.com/wp-content/uploads/2019/08/ob-10a-fig-48-mci-3a.jpgOB 10a Fig 48 MCI 3aFig 48) The intervillous space is packed with mononuclear cells with a rim of cytoplasm (H&E)2019-09-20T19:06:19+00:00weekly0.6https://obstetricalpathology.com/chorioamnionitis/2019-09-08T20:15:38+00:00weekly0.6https://obstetricalpathology.com/preterm-labor-chapter-13-the-cervix-section-a/https://obstetricalpathology.com/wp-content/uploads/2019/09/ob-13a-fig-3.jpgOB 13a Fig 3Fig 3: In another area a more edematous, less cellular matrix is evident. (H&E, 25x)https://obstetricalpathology.com/wp-content/uploads/2019/09/ob-13a-fig-2.jpgOB 13a Fig 2Fig 2: At higher magnification the inflammatory cells appear to be predominantly mononuclear. (H&E 25x)https://obstetricalpathology.com/wp-content/uploads/2019/09/ob-13a-fig-1.jpgOB 13a Fig 1Fig 1: This is a low magnification of a gravid cervix with some myometrial cells to the right, then a denser fibrous area in the middle, and finally an area with loose stroma and many inflammatory cells. (H&E, 4x)2019-09-08T20:13:41+00:00weekly0.6https://obstetricalpathology.com/chap-9a-spiral-arteries/https://obstetricalpathology.com/wp-content/uploads/2019/06/decidua-vera-vasculopathy.jpgDecidua vera vasculopathyhttps://obstetricalpathology.com/wp-content/uploads/2019/06/ob9a-fig39.jpgOB9a fig39Fig 39: This is a high power of the above section showing what appears to be either fibrin or fibrinous necrosis but certainly thinning and dilatation of the artery wall. (H&E 20x)https://obstetricalpathology.com/wp-content/uploads/2019/06/ob9a-fig38.jpgOB9a Fig38Fig 38: This low power image of the membranous decidua sectioned tangential to the membrane plane shows in the convoluted cross sections of a spiral artery, which appears dilated in the right corner of the field. The placenta was uncomplicated with a 3000 kg infant at term with no maternal history of toxemia or autoimmune disease. (H&E, 4X)https://obstetricalpathology.com/wp-content/uploads/2019/06/ob9a-fig37.jpgOB9a Fig37Fig 37: The vessel identified as number 10 demonstrates dilated segments of spiral artery from medial muscle destruction, but this is due to cytotrophoblast that has migrated into the parietal decidua (arrows). (H&E, 10x)https://obstetricalpathology.com/wp-content/uploads/2019/06/ob9a-fig36.jpgOb9a Fig36Fig 36: Similar to the sample in slide 35 there is a spiral artery with a dilated segment. The sample was labeled 10 for identification of the corresponding microscopic sample. https://obstetricalpathology.com/wp-content/uploads/2019/06/ob9a-fig35.jpgOB9a Fig35Fig 35: This is a membrane laid flat demonstrating a spiral artery with focal dilatation and hemorrhage.https://obstetricalpathology.com/wp-content/uploads/2019/06/ob9a-fig34.jpgob9a Fig34Fig 34: This image of placenta demonstrates massive chronic intervillositis with the many monocytic cells stained deeply dark brown for the antigen CD68. The arrow demonstrates the paler, but still positive for CD68 cytotrophoblastic cells embedded in the surrounding fibrinoid. (CD68, 20x)https://obstetricalpathology.com/wp-content/uploads/2019/06/ob9a-fig33.jpgOB9a Fig33Fig 33: A high power frozen section of a spiral artery from the same placenta in Fig 32 stained with Oil Red O to detect fat. Small globules of the fat can be seen in the fibrinoid of the vessel.https://obstetricalpathology.com/wp-content/uploads/2019/06/ob9a-fig32.jpgOB9a fig32Fig 32: The specimen is from the decidua attached to the fetal membranes in a mother with toxemia. There are three cross sections through a spiral artery. The cross section on the right shows a dilated portion with loss of muscular wall. Where the muscular wall would have been there is a thick, pink (eosinophilic), smudged appearance. This is the fibrinoid necrosis. Within this structure, the arrows point to foamy clear macrophages. There are also scattered mononuclear inflammatory cells. https://obstetricalpathology.com/wp-content/uploads/2019/06/ob9a-fig31.jpgOB9a Fig31Fig 31: This is the same placenta showing brown immunostaining with an antibody to platelet endothelial cell adhesion molecule (CD31) that stains endothelial cells in the fetal vessels in the villi, and the lining of a vessel in the decidua, which may be a large vein, but a deeper portion of a spiral artery cannot be excluded. (CD31, 10x)2019-09-08T20:10:23+00:00weekly0.6https://obstetricalpathology.com/placental-infarction/2019-09-08T20:09:38+00:00weekly0.6https://obstetricalpathology.com/fetal-vascular-malperfusion/https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-3-fig-25.jpgchap 3 fig 25Fig 25: The asterisks demonstrate thrombi in a stem vessel in the placenta from the described case. (H&E, 20x)https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-3-fig-24.jpgchap 3 fig 24Fig 24: A smooth margin of pallor from occluded stem vessels in the placenta from marginal compression? (H&E, 20x)https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-3-fig-23.jpgchap 3 fig 23Fig 23: The villi in the center demonstrate lympho-histiocytic villitis with loss of fetal vessels and a mild increase in inflammatory cells. The arrow points to a thrombus in a small vessel. (H&E, 20x)https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-3-fig-22.jpgchap 3 fig 22Fig 22: This stem vessel from a prolonged postmortem intrauterine retention in a twin shows fibroblast ingrowth into the clotted blood. The red cells are not fragmented as in HEV, but this has not been demonstrated to be a reliable feature to distinguish premortem from postmortem fetal vascular obstructive changes. (H&E, 20x)https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-3-fig-20b.jpgchap 3 fig 20bFig 20b: These slices are fixed in formalin and show the pale areas very clearly (*) with one large stem vessel thrombus evident at the arrow.https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-3-fig-20a.jpgchap 3 fig 20aFig 20a: These slices of a placenta demonstrate pale areas that proved to be due to HEV, FVM that likely would not have been as clearly seen with the fresh cut placenta.https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-3-fig-19b.jpgchap 3 fig 19bFig 19b: The surface of this placenta shows a single slightly red stained artery (arrow) compared to the adjacent arteries in an area of FVM.https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-3-fig-19a.jpgchap 3 fig 19aFig 19a: An umbilical artery was injected with blue dyed barium and demonstrates the small area of placental still being perfused. The large red vessels are those involved in FVM. The arrows point to areas of mural thrombi.https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-3-fig-18.jpgchap 3 fig 18Fig 18: The villi in this picture have an eosinophilic (pink) stroma and absent capillaries. The small villi in the center show blue lines (ferrugination) just below the surface that is due to the deposition of iron transported by the syncytiotrophoblast to the stroma in the absence of circulatory transport back to the fetus. (H&E, 20x)https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-3-fig-17.jpgchap 3 fig 17Fig 17: This even higher power of villi from the same area as figure 16 shows the blue nuclear fragments of karyorrhexis and fragmented red blood cells. (H&E, 40x)2019-09-08T20:07:41+00:00weekly0.6https://obstetricalpathology.com/breus-mole-subchorionic-thrombohematoma-fibrin-plaques-and-subchorionic-cysts/https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-37b.jpgChap 4 fig 37bFig 15b This photomicrograph demonstrates a small cyst in a septum in the middle of the intervillous space. (H&E, 10x)https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-37a.jpgChap 4 Fig 37aFig 15a This photomicrograph shows multiple chorionic microcysts (arrows) in the chorion layer of the fetal membranes. (H&E, 4x)https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-36b.jpgChap 4 Fig 36bFig 14b The fetal surface of this placenta has a cyst that appears to arise from the partial circumvallate margin. https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-36a.jpgChap 4 Fig 36aFig 14a The fetal surface demonstrates a large, drooping, thin walled chorionic cyst (arrows) that has a base at a fibrin plaque (“fibrin”).https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-35a.jpgChap 4 Fig 35aFig 13 The fetal surface of this placenta shows a large, multilobular chorionic cysts filled with dark blood stained fluid. There is also a partial circumvallate margin of the placenta (c).https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-34d.jpgChap 4 Fig 34dFig 12d This photomicrograph shows the expansion of a small surface chorionic cyst with fluid. (H&E, 4x)https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-34c.jpgChap 4 Fig 34cFig 12c This photomicrograph shows a larger volume of entrapped cytotrophoblast in the subchorionic fibrinoid that have formed a pseudohttps://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-34b.jpgChap 4 Fig 34bFig 12b The fetal surface of the placenta is at the top of the photomicrograph with the pink subchorionic fibrinoid running diagonally above the intervillous space. The arrow points to some trapped residual cytotrophoblast. (H&E, 20x)https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-34a.jpgChap 4 Fig 34aFig 12a This is a photomicrograph of the fetal surface of an immature placenta showing the thick layer of cytotrophoblastic epithelium lining the top of the intervillous space (arrow). (H&E, 4x)https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-33b.jpgChap 4 Fig 33bFig 11b The fetal surface shows a large chorionic cyst with some hemorrhage into it. 2019-09-08T20:06:45+00:00weekly0.6https://obstetricalpathology.com/placenta-accreta/https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-8.jpgChap 4 Fig 8Fig 8: The base of this placenta shows an area of incidental attached myometrium (arrow). (H&E, 10x)https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-7e.jpgChap 4 Fig 7eFig 7e: This is the normal basal plate of the placenta with the cytotrophoblast (arrow) implanting on the pale cells of the decidualized endometrial stroma. (H&E, 20x)https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-7d.jpgChap 4 Fig 7dFig 7d: This microphotograph is from the base of the placenta accreta in which the placenta was removed from the uterus before hysteretomy. The base of the placenta sits directly on the smooth muscle of the myometrium (arrow). (H&E, 4x)https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-7c.jpgChap 4 Fig 7cFig 7c: This high power demonstrates the cytotrophblast cells (arrow points to a typical cell) of the chorionic plate embedding on and infiltrating into the very pink (eosinophilic) smooth muscle cells of the myometrium in a placenta accrete uterus. (H&E, 20x)https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-7b.jpgChap 4 Fig 7bFig 7b: The placenta in this image has a basal chorion implanted directly on a layer of serosa. This is the same placenta as in Fig 5d-e. (H&E, 20x)https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-7a.jpgChap 4 Fig 7aFig 7a: This is a low magnification of the placenta on top with a thick chorionic base sitting over a relatively thin myometrium that ends at the serosa. This is from the same placenta as in Fig 5a-c. (H&E)https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-6e.jpgChap 4 Fig 6eFig 6e: This close up photograph better demonstrates the placenta previa portion of this placental accreta.https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-6d.jpgChap 4 Fig 6dFig 6d: This bisected uterus shows an attached placenta over the lower uterine segment that is also over the internal os of the cervix (placenta previa).https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-6c.jpgChap 4 Fig 6cFig 6c: The same uterus in cross section demonstrates the completely attached placenta. The myometrium is pale and the placenta red.https://obstetricalpathology.com/wp-content/uploads/2018/05/chap-4-fig-6b.jpgChap 4 Fig 6bFig 6b: The incised uterus demonstrates adherent placenta on almost the entire uterine surface (placenta membranacea). There was no decidua over the uterine surface.2019-09-08T20:05:05+00:00weekly0.6https://obstetricalpathology.com/subacute-necrotizing-funisitis-snf/https://obstetricalpathology.com/wp-content/uploads/2018/04/fig-11c-snf.jpgFig 11c SNFFig 11c) Silver stain demonstrating a spirochete (arrow). They were through out Wharton's Jelly.https://obstetricalpathology.com/wp-content/uploads/2018/04/fig-11b-snf.jpgFig 11b SNFFig 11b) Higher power demonstrating chronic inflammation in the umbilical vein media.https://obstetricalpathology.com/wp-content/uploads/2018/04/fig-11a-snf.jpgFig 11a SNFFig 11a) SNF lesion in a stillborn infant with syphilis.https://obstetricalpathology.com/wp-content/uploads/2018/04/fig-10d-snf.jpgFig 10d SNFFig 10d) An anti-HSV immuno-stain demonstrates that the cells in Wharton's Jelly had become infected with HSV.https://obstetricalpathology.com/wp-content/uploads/2018/04/fig-10c-snf.jpgFIg 10c SNFFig 10c) In some areas there appear to be plasma cells.https://obstetricalpathology.com/wp-content/uploads/2018/04/fig-10b-snf.jpgFig 10b SNFFig 10b) Just beneath the surface there is a chronic inflammatory infiltration and necrotic cells.https://obstetricalpathology.com/wp-content/uploads/2018/04/fig-10a-snf.jpgFig 10a SNFFig 10a) The umbilical cord shows minimal findings of SNF.https://obstetricalpathology.com/wp-content/uploads/2018/04/fig-9e-snf.jpgFIg 9e SNFFig 9e) Silver stain showing the invasive pseudohyphae of candida invading into Wharton's Jelly.https://obstetricalpathology.com/wp-content/uploads/2018/04/fig-9d-snf.jpgFig 9d SNFFig 9d) High power shows neutrophils and necrotic debris on the right, and slightly basophilic hyphae on the left.https://obstetricalpathology.com/wp-content/uploads/2018/04/fig-9c-snf.jpgFig 9c SNFFig 9 c) Medium power of the same lesion.2019-09-08T19:57:01+00:00weekly0.6https://obstetricalpathology.com/maternal-floor-infarction-massive-fibrinoid-infiltration-mfi/https://obstetricalpathology.com/wp-content/uploads/2018/04/mfifig20d.jpgmfifig20dFig 20dhttps://obstetricalpathology.com/wp-content/uploads/2018/04/mfifig20c.jpgmfifig20cFig 20chttps://obstetricalpathology.com/wp-content/uploads/2018/04/mfifig20b.jpgmfifig20bFig 20bhttps://obstetricalpathology.com/wp-content/uploads/2018/04/mfifig20a.jpgmfifig20aFig 20ahttps://obstetricalpathology.com/wp-content/uploads/2018/04/mfifig19.jpgmfifig19Fig 19https://obstetricalpathology.com/wp-content/uploads/2018/04/mfifig19g.jpgmfifig19gFig 18ghttps://obstetricalpathology.com/wp-content/uploads/2018/04/mfifig18f.jpgmfifig18fFig 18fhttps://obstetricalpathology.com/wp-content/uploads/2018/04/mfifig18e.jpgmfifig18eFig 18ehttps://obstetricalpathology.com/wp-content/uploads/2018/04/mfifig18d.jpgmfifig18dFig 18dhttps://obstetricalpathology.com/wp-content/uploads/2018/04/mfifig18c.jpgmfifig18cFig 18c2019-09-08T19:55:48+00:00weekly0.6https://obstetricalpathology.com/short-case-report-3-umbilical-cord-prolapse/https://obstetricalpathology.com/wp-content/uploads/2018/03/scr2-fig-2.jpgSCR2 fig 2Fig 2: The arrow points to the pericardial effusion.https://obstetricalpathology.com/wp-content/uploads/2018/03/scr2-fig-1-e1520965908403.jpgSCR2 fig 1Fig 1: There is a relatively sharp demarcation between the white portion and the reddened portion of the umbilical cord.2019-09-08T19:53:01+00:00weekly0.6https://obstetricalpathology.com/short-case-report-4-multiple-nuchal-cord-wrappings/https://obstetricalpathology.com/wp-content/uploads/2018/03/scr-fig-8.jpgSCR fig 8Fig 8) The arrow points to squames from aspirated vernix and meconium. The pigment does not display well. https://obstetricalpathology.com/wp-content/uploads/2018/03/scr4-fig-71.jpgSCR4 fig 7Fig 7) The arrow points to a petechial hemorrhage in the thymus.https://obstetricalpathology.com/wp-content/uploads/2018/03/scr4-fig-7.jpgSCR4 fig 7https://obstetricalpathology.com/wp-content/uploads/2018/03/scr4-fig-6.jpgSCR4 fig 6Fig 6) There were increased nucleated red blood cells seen here in the umbilical vein.https://obstetricalpathology.com/wp-content/uploads/2018/03/scr4-fig5.jpgSCR4 fig5Fig 5) This section of chorion shows the absence of neutrophils. There are meconium macrophages but they do not display well.https://obstetricalpathology.com/wp-content/uploads/2018/03/scr4fig4.jpgSCR4fig4Fig 4) There are sparse subintimal neutrophils in the umbilical arteryhttps://obstetricalpathology.com/wp-content/uploads/2018/03/scr4fig-3.jpgSCR4fig 3Fig 3) This even later tracing shows progressive bradycardia.https://obstetricalpathology.com/wp-content/uploads/2018/03/scr4fig2.jpgSCR4fig2Fig 2) This later tracing shows 2 long deep variable decelerations, and loss of normal beat-to-beat variability.https://obstetricalpathology.com/wp-content/uploads/2018/03/scr4-fig-1.jpgSCR4 fig 1Fig 1) This fetal heart rate tracing on the right side shows a deep variable deceleration. The beat to beat variability is normal.2019-09-08T19:52:28+00:00weekly0.6https://obstetricalpathology.com/short-case-review-2-umbilical-cord-compression/https://obstetricalpathology.com/wp-content/uploads/2018/03/sc1fig5.jpgsc1fig5https://obstetricalpathology.com/wp-content/uploads/2018/03/sc1fig4.jpgsc1fig4https://obstetricalpathology.com/wp-content/uploads/2018/03/sc1fig3.jpgsc1fig3https://obstetricalpathology.com/wp-content/uploads/2018/03/sc1fig2.jpgsc1fig2https://obstetricalpathology.com/wp-content/uploads/2018/03/sc1fig1.jpgsc1fig12019-09-08T19:50:41+00:00weekly0.6https://obstetricalpathology.com/short-case-review-9-1/https://obstetricalpathology.com/wp-content/uploads/2016/01/slide41.jpgSlide4https://obstetricalpathology.com/wp-content/uploads/2016/01/slide33.jpgSlide3https://obstetricalpathology.com/wp-content/uploads/2016/01/slide23.jpgSlide2https://obstetricalpathology.com/wp-content/uploads/2016/01/slide14.jpgSlide12019-09-08T19:49:58+00:00weekly0.6https://obstetricalpathology.com/umbilical-cord-accident-part-2/https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-2-fig-21b.pngchap 2 fig 21bFigure 21b: This photomicrograph of Wharton’s jelly shows mast cells identifiable by immunological markers using that cause brown peroxidase precipitate. (CD117 immunohistochemistry, 40x) https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-2-fig-21a.pngchap 2 fig 21aFigure 21a: This photomicrograph of Wharton’s jelly shows mast cells identifiable by the metachromasia that makes them appear purple with a blue dye. (Giemsa, 40x)https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-2-fig-20b.pngchap 2 fig 20bFigure 20b: This direct photograph of a microscope slide demonstrates a cross section of an umbilical cord from a stillborn infant demonstrating unconstricted umbilical arteries with open clear spaces in the lumens. The arteries may never have constricted normally or they may have dilated with death of the smooth muscle cells in the artery wall.https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-2-fig-20a.pngchap 2 fig 20aFigure 20a: This direct photograph of a microscope slide demonstrates the normal cross section of a delivered umbilical cord with two constricted arteries and open vein.https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-2-fig-19.pngchap 2 fig 19Figure 19: This close up of the umbilicus in a mid trimester stillborn infant shows the twisting and narrowing of the cord insertion often found with prolonged intrauterine postmortem retention in this gestational range.https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-2-fig-18.pngchap 2 fig 18Figure 18: This furcate (fork like) umbilical cord insertion demonstrates the loss of Wharton’s jelly that could make the vessels subject to collapse with the application of fetal turning. The infant was stillborn without other explanation.https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-2-fig-17c.pngchap 2 fig 17cFigure 17c: This marginal insertion of the cord in a stillborn infant appeared narrowed and vulnerable to twisting.https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-2-fig-17b.pngchap 2 fig 17bFigure 17b: This marginal insertion of the cord in a stillborn infant appeared narrowed and eroded.https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-2-fig-17a.pngchap 2 fig 17aFigure 17a: The umbilical cord is inserted into the margin of the placenta and did not cause any known complications.https://obstetricalpathology.com/wp-content/uploads/2018/08/chap-2-fig-16c.pngchap 2 fig 16cFigure 16c: In this early gestation fetus delivered within a decidual cast, there appears to be superimposed twists on the umbilical vascular coil likely due to postmortem rotation of the fetus.2019-09-08T19:49:05+00:00weekly0.6https://obstetricalpathology.com/meconium/https://obstetricalpathology.com/wp-content/uploads/2016/01/slide32.jpgSlide3https://obstetricalpathology.com/wp-content/uploads/2016/01/slide22.jpgSlide2https://obstetricalpathology.com/wp-content/uploads/2016/01/slide13.jpgSlide12019-09-08T19:47:10+00:00weekly0.6https://obstetricalpathology.com/stillbirth-asphyxia/https://obstetricalpathology.com/wp-content/uploads/2016/01/slide31.jpgSlide3https://obstetricalpathology.com/wp-content/uploads/2016/01/slide21.jpgSlide2https://obstetricalpathology.com/wp-content/uploads/2016/01/slide12.jpgSlide12019-09-08T19:46:15+00:00weekly0.6https://obstetricalpathology.com/fetal-asphyxia-introduction-and-john-little/https://obstetricalpathology.com/wp-content/uploads/2016/01/slide61.jpgSlide6https://obstetricalpathology.com/wp-content/uploads/2016/01/slide6.jpgSlide6https://obstetricalpathology.com/wp-content/uploads/2016/01/slide5.jpgSlide5https://obstetricalpathology.com/wp-content/uploads/2016/01/slide4.jpgSlide4https://obstetricalpathology.com/wp-content/uploads/2016/01/slide3.jpgSlide3https://obstetricalpathology.com/wp-content/uploads/2016/01/slide2.jpgSlide2https://obstetricalpathology.com/wp-content/uploads/2016/01/slide1.jpgSlide1https://obstetricalpathology.com/wp-content/uploads/2016/01/slide8.jpgSlide8https://obstetricalpathology.com/wp-content/uploads/2016/01/slide7.jpgSlide7https://obstetricalpathology.com/wp-content/uploads/2015/12/image002.pngimage0022019-09-08T19:45:17+00:00weekly0.6https://obstetricalpathology.com/ovine-model/2019-09-08T19:44:48+00:00weekly0.6https://obstetricalpathology.com/dr-ron-myers/2019-09-08T19:44:08+00:00weekly0.6https://obstetricalpathology.com/primate-model/https://obstetricalpathology.com/wp-content/uploads/2016/01/slide11.jpgSlide12019-09-08T19:43:08+00:00weekly0.6https://obstetricalpathology.com/guinea-pigs/2019-09-08T19:41:35+00:00weekly0.6https://obstetricalpathology.com/twilight-sleep/2019-09-08T19:40:19+00:00weekly0.6https://obstetricalpathology.com/2019/08/27/a-new-page-on-villitis-of-unknown-etiology/2019-08-27T23:46:53+00:00monthlyhttps://obstetricalpathology.com/2019/06/27/comments-on-spiral-artery/2019-06-27T15:38:46+00:00monthlyhttps://obstetricalpathology.com/2019/02/08/an-obstetrical-care-research-center/2019-02-08T17:50:57+00:00monthlyhttps://obstetricalpathology.com/2018/10/18/new-page-on-fetal-cells/2018-10-18T14:40:33+00:00monthlyhttps://obstetricalpathology.com/chapter-7-fetal-blood-cells/https://obstetricalpathology.com/wp-content/uploads/2018/10/chap-7-fig-3b.jpgChap 7 Fig 3BFig 3B: The image is an umbilical vein from a term, stillborn infant of a diabetic mother. The arrows point to nRBCs. (H&E 20x)https://obstetricalpathology.com/wp-content/uploads/2018/10/chap-7-fig-3a.jpgChap 7 Fig 3AFig 3A: The image is of a stem vessel in the placenta of a term stillborn infant with marked pallor. The arrows point to typical nRBS with very round dense nuclei and a pale pink hemoglobin cytoplasmic halo. Because of overlapping and partial cells, a relative count of nRBS compared to WBC is difficult, but can not identify increased nRBCs. (H&E 20x)https://obstetricalpathology.com/wp-content/uploads/2018/10/chap-7-fig-2b.jpgChap 7 Fig 2BFig 2 B: Liver from a term stillborn infant with erythroblastosis demonstrating the increased in size, and synchrony of erythroblastic cells in the liver. (H&E, 20x) https://obstetricalpathology.com/wp-content/uploads/2018/10/chap-7-fig-2a.jpgChap 7 Fig 2AFig 2A: Liver from a term infant dying following asphyxia at birth. The large arrow points to a focus of sinusoidal erythropoiesis. The ** show an area of cell necrosis. The small arrow points to a nucleated red cell that may be in the circulation. (H&E, 100X)https://obstetricalpathology.com/wp-content/uploads/2018/10/chap-7-fig-1c.jpgchap 7 Fig 1CFig 1C: The arrow points to a nucleated erythrocyte precursor with a central pale eosinophilic inclusion of Parvo virus. (H&E 100x)https://obstetricalpathology.com/wp-content/uploads/2018/10/chap-7-fig-1b.jpgchap 7 Fig 1BFig 1B: The arrow points to a cell with CMV nuclear and cytoplasmic inclusions in the same placenta as Fig 1A. The dotted arrow points to deposition of hemosiderin which is characteristic of CMV infection in the placenta. (H&E 40x)https://obstetricalpathology.com/wp-content/uploads/2018/10/chap-7-fig-1a.jpgChap 7 Fig 1AFig 1A: The arrows demonstrate increased nucleated red cells in the fetal capillaries in the placental villi in an infant with congenital CMV infection. (H&E 20x)2018-10-18T14:34:16+00:00weekly0.6https://obstetricalpathology.com/2018/09/11/tipps-the-international-placental-pathology-study-group/2018-09-13T14:53:41+00:00monthlyhttps://obstetricalpathology.com/2018/09/08/a-simple-measure-kick-counts-and-a-plea-for-measuring-the-untethered-length-of-the-umbilical-cord/2018-09-08T17:01:41+00:00monthlyhttps://obstetricalpathology.com/autopsy-manual-measurements-and-weights/2018-08-27T15:22:17+00:00weekly0.6https://obstetricalpathology.com/2018/08/24/umbilical-cord-accident-2/2018-08-24T19:30:30+00:00monthlyhttps://obstetricalpathology.com/2018/08/23/just-added-page-on-fetal-vascular-malperfusion/2018-08-23T14:21:55+00:00monthlyhttps://obstetricalpathology.com/autopsy-manual-in-utero-lie-and-presentation/https://obstetricalpathology.com/wp-content/uploads/2018/08/slide08.jpgSlide08https://obstetricalpathology.com/wp-content/uploads/2018/08/slide07.jpgSlide07https://obstetricalpathology.com/wp-content/uploads/2018/08/slide06.jpgSlide06https://obstetricalpathology.com/wp-content/uploads/2018/08/slide05.jpgSlide052018-08-21T01:57:11+00:00weekly0.6https://obstetricalpathology.com/pathology-manual-gestational-age/https://obstetricalpathology.com/wp-content/uploads/2018/08/slide19.jpgSlide19https://obstetricalpathology.com/wp-content/uploads/2018/08/slide32.jpgSlide32https://obstetricalpathology.com/wp-content/uploads/2018/08/slide26.jpgSlide26https://obstetricalpathology.com/wp-content/uploads/2018/08/slide31.jpgSlide31https://obstetricalpathology.com/wp-content/uploads/2018/08/slide30.jpgSlide30https://obstetricalpathology.com/wp-content/uploads/2018/08/slide29.jpgSlide29https://obstetricalpathology.com/wp-content/uploads/2018/08/slide28.jpgSlide28https://obstetricalpathology.com/wp-content/uploads/2018/08/slide27.jpgSlide27https://obstetricalpathology.com/wp-content/uploads/2018/08/slide25.jpgSlide25https://obstetricalpathology.com/wp-content/uploads/2018/08/slide241.jpgSlide242018-08-19T15:30:19+00:00weekly0.6https://obstetricalpathology.com/duration-of-postmortem-retention/https://obstetricalpathology.com/wp-content/uploads/2018/07/ca006740xli.jpgCA006740xLiThe hepatocytes nuclei are have lost basophilia. There is some basophilia in hematopoietic nuclei.https://obstetricalpathology.com/wp-content/uploads/2018/07/ca006725xkid.jpgCA006725xkidFig 2 Renal tubules show greater than 1% loss of nuclear basophilia.https://obstetricalpathology.com/wp-content/uploads/2018/07/slide7.jpgSlide72018-08-19T15:29:39+00:00weekly0.6https://obstetricalpathology.com/2018/08/19/pathology-manual/2018-08-19T15:28:21+00:00monthlyhttps://obstetricalpathology.com/2018/08/16/stillbirth-research-idea/2018-08-17T01:31:41+00:00monthlyhttps://obstetricalpathology.com/research-idea-for-evaluation-of-stillbirth-bob-bendon-m-d/2018-08-17T01:03:37+00:00weekly0.6https://obstetricalpathology.com/2018/07/18/open-letter-to-obstetricians/2018-07-18T16:15:39+00:00monthlyhttps://obstetricalpathology.com/an-open-letter-to-obstetricians-about-fetal-asphyxia-from-a-pathologists-perspective/2018-07-18T16:09:18+00:00weekly0.6https://obstetricalpathology.com/2018/07/11/postmortem-retention-etc/2018-07-11T17:00:46+00:00monthlyhttps://obstetricalpathology.com/2018/05/30/breus-mole-etc/2018-05-30T20:11:57+00:00monthlyhttps://obstetricalpathology.com/2018/05/20/placenta-accreta-post/2018-05-20T20:34:39+00:00monthlyhttps://obstetricalpathology.com/2018/04/20/posting-on-snf/2018-04-20T18:39:06+00:00monthlyhttps://obstetricalpathology.com/2018/04/02/mfi-page-added/2018-04-02T19:34:02+00:00monthlyhttps://obstetricalpathology.com/2018/03/14/short-case-reports/https://obstetricalpathology.com/wp-content/uploads/2018/03/cs15-5509_10xstem.jpgCS15-5509_10xstemThis stem villus demonstrates only recent in growth of endothelial cells from post mortem retention.https://obstetricalpathology.com/wp-content/uploads/2018/03/cs15-5509_10xhev.jpgCS15-5509_10xhevFig 1) These vilil show classic findings of HEV with an occluded lumen and extrusion of red cells and fragments.2018-03-14T16:11:10+00:00monthlyhttps://obstetricalpathology.com/2018/03/04/short-cord-due-to-cord-wrapping/https://obstetricalpathology.com/wp-content/uploads/2018/03/save0002.jpgSAVE0002Fig 2: PVC pipeshttps://obstetricalpathology.com/wp-content/uploads/2018/03/save0003.jpgSAVE0003Fig 1: Our equipment2018-03-04T14:56:25+00:00monthlyhttps://obstetricalpathology.com/2018/02/24/multiple-placental-infarctions/https://obstetricalpathology.com/wp-content/uploads/2018/02/blog-fig-3.jpgblog 3FIG 3https://obstetricalpathology.com/wp-content/uploads/2018/02/fig-blog-1b.jpgfig 1bhttps://obstetricalpathology.com/wp-content/uploads/2018/02/11.jpgFIG 2https://obstetricalpathology.com/wp-content/uploads/2018/02/12.jpg12Fig 2: Multiple acute infarctions 2018-02-24T15:15:24+00:00monthlyhttps://obstetricalpathology.com/2018/02/14/restarting-blog/2018-02-14T18:51:22+00:00monthlyhttps://obstetricalpathology.com/fetal-asphyxia-a-pathologists-perspective/https://obstetricalpathology.com/wp-content/uploads/2018/02/bendon-fig-6.jpgBendon Fig 6Figure 6https://obstetricalpathology.com/wp-content/uploads/2018/02/bendon-fig-5.jpgBendon FIg 5Figure 5https://obstetricalpathology.com/wp-content/uploads/2018/02/bendon-fig-4.jpgBendon Fig 4Figure 4https://obstetricalpathology.com/wp-content/uploads/2018/02/bendon-fig-3.jpgBendon Fig 3Figure 3https://obstetricalpathology.com/wp-content/uploads/2018/02/bendon-fig-2.jpgBendon Fig 2Figure 2https://obstetricalpathology.com/wp-content/uploads/2018/02/bendon-fig-1.jpgBendon Fig 1Figure 12018-02-14T18:47:01+00:00weekly0.6https://obstetricalpathology.com/2016/07/23/idm-case-6-intermittent-asphyxia-of-unknown-cause/https://obstetricalpathology.com/wp-content/uploads/2016/07/weigths-ca10-52.jpgweigths CA10-52https://obstetricalpathology.com/wp-content/uploads/2016/07/fig-5-ca10-52_40xhippo.jpgFig 5 CA10-52_40xhippohttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-4-ca10-52_20xlung.jpgFig 4 CA10-52_20xlunghttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-3-ca10-52_20xliv.jpgFig 3 CA10-52_20xlivhttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-2-ca10-52.jpgFig 2 CA10-52https://obstetricalpathology.com/wp-content/uploads/2016/07/fig-1-ca10-52_2xskin.jpgFig 1 CA10-52_2xskin2016-07-23T13:37:08+00:00monthlyhttps://obstetricalpathology.com/2016/07/22/idm-case-5-pulmonary-congestion-and-hemorrhage/https://obstetricalpathology.com/wp-content/uploads/2016/07/slide14.jpgSlide1https://obstetricalpathology.com/wp-content/uploads/2016/07/fig-7-ca00-65_40xlung2.jpgFig 7 CA00-65_40xlung2https://obstetricalpathology.com/wp-content/uploads/2016/07/fig-6-ca00-65_20xlung.jpgFig 6 CA00-65_20xlunghttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-5-ca00-65_2xlung.jpgFig 5 CA00-65_2xlunghttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-4-ca00-65_10xplac.jpgFig 4 CA00-65_10xplachttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-3-ca00-65_20xkid.jpgFig 3 CA00-65_20xkidhttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-2-ca00-65_10xadr.jpgFig 2 CA00-65_10xadrhttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-1-ca00-65_2xskin.jpgFig 1 CA00-65_2xskin2016-07-22T14:55:12+00:00monthlyhttps://obstetricalpathology.com/2016/07/21/idm-case4-probably-not-diabetes-related/https://obstetricalpathology.com/wp-content/uploads/2016/07/slide13.jpgSlide1https://obstetricalpathology.com/wp-content/uploads/2016/07/fig-8-ca02-94_40xvillinrbc.jpgFig 8 CA02-94_40xvillinrbchttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-6-ca02-94_20xlung.jpgFig 6 CA02-94_20xlunghttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-5-ca02-94_40xmembr.jpgFig 5 CA02-94_40xmembrhttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-4-ca02-94_20xsciv.jpgFig 4 CA02-94_20xscivhttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-3-ca02-94_20xvilli-vasc.jpgFig 3 CA02-94_20xvilli vaschttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-2-ca02-94_20xadr.jpgFig 2 CA02-94_20xadrhttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-1-ca02-94_20xkid.jpgFig 1 CA02-94_20xkid2016-07-21T14:29:33+00:00monthlyhttps://obstetricalpathology.com/2016/07/19/idm-case-3-acute-asphyxia-furcate-cord-insertion/https://obstetricalpathology.com/wp-content/uploads/2016/07/weights.jpgweightshttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-6-ca01-22_20xuc.jpgfig 6 CA01-22_20xuchttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-5-ca01-22_10xlung.jpgfig 5 CA01-22_10xlunghttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-4-ca01-22_4xthym.jpgfig 4 CA01-22_4xthymhttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-3-ca01-22_40xpancr.jpgfig 3 CA01-22_40xpancrhttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-2-ca01-22_20xliv.jpgfig 2 CA01-22_20xlivhttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-1.jpgfig 12016-07-19T19:58:33+00:00monthlyhttps://obstetricalpathology.com/2016/07/09/idm-case-2-cord-wrapping-placental-infarctions/https://obstetricalpathology.com/wp-content/uploads/2016/07/fig-11-ca98-26_4xthym.jpgfig 11 CA98-26_4xthymhttps://obstetricalpathology.com/wp-content/uploads/2016/07/slide12.jpgSlide1https://obstetricalpathology.com/wp-content/uploads/2016/07/fig-9ca98-26_2xkid.jpgfig 9CA98-26_2xkidhttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-8-ca98-26_20xavasc.jpgfig 8 CA98-26_20xavaschttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-7-ca98-26_20x-villi.jpgfig 7 CA98-26_20x villihttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-6-ca98-26_20x-lung.jpgfig 6 CA98-26_20x lunghttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-5-ca98-26_40x-membr.jpgfig 5 CA98-26_40x membrhttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-4ca98-26_40xpons.jpgfig 4CA98-26_40xponshttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-3-ca98-26_40xhippo.jpgFig 3 CA98-26_40xhippohttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-2-ca98-26_2xplac.jpgfig 2 CA98-26_2xplac2016-07-09T14:44:14+00:00monthlyhttps://obstetricalpathology.com/2016/07/07/idm-case-1-diabetes-and-hypothyroidism/https://obstetricalpathology.com/wp-content/uploads/2016/07/slide11.jpgSlide1https://obstetricalpathology.com/wp-content/uploads/2016/07/fig-9-ka14-13_10xthym.jpgFig 9 KA14-13_10xthymhttps://obstetricalpathology.com/wp-content/uploads/2016/07/slide4.jpgSlide4https://obstetricalpathology.com/wp-content/uploads/2016/07/fig-7-ka14-13_40xplac.jpgFig 7 KA14-13_40xplachttps://obstetricalpathology.com/wp-content/uploads/2016/07/slide3.jpgSlide3https://obstetricalpathology.com/wp-content/uploads/2016/07/slide1.jpgSlide1https://obstetricalpathology.com/wp-content/uploads/2016/07/slide2.jpgSlide2https://obstetricalpathology.com/wp-content/uploads/2016/07/fig-3-ka14-13_2xnob.jpgFig 3 KA14-13_2xnobhttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-2-ka14-13_20xbronch.jpgFig 2 KA14-13_20xbronchhttps://obstetricalpathology.com/wp-content/uploads/2016/07/fig-1-ka14-13_20xkid.jpgFig 1 KA14-13_20xkid2016-07-07T15:18:49+00:00monthlyhttps://obstetricalpathology.com/2016/07/06/autopsies-of-stillborn-infants-of-diabetic-mothers/2016-07-06T14:15:55+00:00monthlyhttps://obstetricalpathology.com/2016/03/29/placental-lesions-in-stillborn-small-for-gestational-age-infants/https://obstetricalpathology.com/wp-content/uploads/2016/03/presentation5.jpgPresentation5https://obstetricalpathology.com/wp-content/uploads/2016/03/presentation4.jpgPresentation4https://obstetricalpathology.com/wp-content/uploads/2016/03/presentation3.jpgPresentation3https://obstetricalpathology.com/wp-content/uploads/2016/03/presentation2.jpgPresentation2https://obstetricalpathology.com/wp-content/uploads/2016/03/presentation1.jpgPresentation12016-07-06T13:46:09+00:00monthlyhttps://obstetricalpathology.com/2016/03/14/material-from-chicago-course-on-upi/https://obstetricalpathology.com/wp-content/uploads/2016/03/30.jpg30https://obstetricalpathology.com/wp-content/uploads/2016/03/29.jpg29https://obstetricalpathology.com/wp-content/uploads/2016/03/21.jpg21https://obstetricalpathology.com/wp-content/uploads/2016/03/20.jpg20https://obstetricalpathology.com/wp-content/uploads/2016/03/19.jpg19https://obstetricalpathology.com/wp-content/uploads/2016/03/15.jpg15https://obstetricalpathology.com/wp-content/uploads/2016/03/14.jpg14https://obstetricalpathology.com/wp-content/uploads/2016/03/16.jpg16https://obstetricalpathology.com/wp-content/uploads/2016/03/8.jpg8https://obstetricalpathology.com/wp-content/uploads/2016/03/5.jpg52016-03-14T18:06:45+00:00monthlyhttps://obstetricalpathology.com/2016/02/23/subutex-modification-of-placental-maturation/https://obstetricalpathology.com/wp-content/uploads/2016/02/slide5.jpgSlide5https://obstetricalpathology.com/wp-content/uploads/2016/02/slide4.jpgSlide4https://obstetricalpathology.com/wp-content/uploads/2016/02/slide3.jpgSlide3https://obstetricalpathology.com/wp-content/uploads/2016/02/slide2.jpgSlide2https://obstetricalpathology.com/wp-content/uploads/2016/02/slide1.jpgSlide12016-02-23T17:17:16+00:00monthlyhttps://obstetricalpathology.com/2016/01/26/a-study-of-stillbirth/2016-01-26T15:59:09+00:00monthlyhttps://obstetricalpathology.com/2016/01/13/looking-at-fetal-blood-in-the-placenta/https://obstetricalpathology.com/wp-content/uploads/2016/01/10102.jpg10102Slightly less crowded together acanthocytes.https://obstetricalpathology.com/wp-content/uploads/2016/01/10098.jpg10098The acanthocytes sometimes appear as knobs or as darker small spheres.https://obstetricalpathology.com/wp-content/uploads/2016/01/triploid-coulter.pngtriploid coulterOn this Coulter strip, the red cell population on the left from the PUBS shows the large cells. The comparison on the right is after a neonatal red cell transfusion which show the normal peak and the residual triploid population.https://obstetricalpathology.com/wp-content/uploads/2016/01/11293.jpg11293The red cells are consistently larger in the fetal vessels compared to the maternal cells in the intervillous space in this 20 week fetus with dysgenic triploidy.https://obstetricalpathology.com/wp-content/uploads/2016/01/parvo.pngparvoThe eosinophilic inclusions of parvo virus in the nucleated red cells.https://obstetricalpathology.com/wp-content/uploads/2016/01/blog-3.pngBlog 3This placenta had scattered possible blasts and an abnormal blood picture but my notes are missing.https://obstetricalpathology.com/wp-content/uploads/2016/01/11287.jpg11287High power showing the leukemic blasts.https://obstetricalpathology.com/wp-content/uploads/2016/01/11289.jpg11289Medium power showing leukemic cells in vessels but also in the villous stroma.https://obstetricalpathology.com/wp-content/uploads/2016/01/11276.jpg11276The fetal blood at first looked like increased nucleated red cells, but the cytoplasm was clear not red, and the overall impression was a monomorphic mononuclear population.https://obstetricalpathology.com/wp-content/uploads/2016/01/11275.jpg11275This low power image shows the increased nucleated blood cells in the stem vessel.2016-01-25T21:14:21+00:00monthlyhttps://obstetricalpathology.com/2013/02/22/more-on-the-adrenal-and-three-ideas/https://obstetricalpathology.com/wp-content/uploads/2013/02/ca99-3_40xtest.jpgTestesInvolution of most Leydig cells. 40xhttps://obstetricalpathology.com/wp-content/uploads/2013/02/ca99-3_40xadr1.jpgAdrenalEarly evidence of involution of the fetal zone 40x2013-02-22T18:09:11+00:00monthlyhttps://obstetricalpathology.com/2013/01/21/stillborn-autopsy-case-2/https://obstetricalpathology.com/wp-content/uploads/2013/01/ca1252spl.jpgca1252splSpleen, left H&E 20x, right iron stain 40s showing pale hemosiderin macrophages.https://obstetricalpathology.com/wp-content/uploads/2013/01/ca12-52_20xkida.jpgCA12-52_20xkidaKidney showing hematopoiesis around a vessel, H&E 20xhttps://obstetricalpathology.com/wp-content/uploads/2013/01/wt-table.jpgwt tablehttps://obstetricalpathology.com/wp-content/uploads/2013/01/ca52-1.jpgCA52 (1)https://obstetricalpathology.com/wp-content/uploads/2013/01/ca12-52_20xplac.jpgCA12-52_20xplac2013-01-21T18:39:01+00:00monthlyhttps://obstetricalpathology.com/2013/01/16/why-do-primates-have-a-fetal-adrenal/2013-01-16T20:20:12+00:00monthlyhttps://obstetricalpathology.com/2012/12/11/chorangiosis/https://obstetricalpathology.com/wp-content/uploads/2012/12/amscope-mt5-2-e1355252461162.jpgFigure 7Dissecting scope of same hyerpvascular villihttps://obstetricalpathology.com/wp-content/uploads/2012/12/cs12-7014_4xplac.jpgfig 5Diffuse increased vascularityhttps://obstetricalpathology.com/wp-content/uploads/2012/12/cs12-7014_20xplac.jpgFigure 6Diffuse increased vascularityhttps://obstetricalpathology.com/wp-content/uploads/2012/12/cs12-7619_10xplac1.jpgfig 4Normal stem villus with attached branch showing capillary proliferationhttps://obstetricalpathology.com/wp-content/uploads/2012/12/cs12-7619_4xplac.jpgCS12-7619_4xplachttps://obstetricalpathology.com/wp-content/uploads/2012/12/cs12-6995_10xvue.jpgFigure 3Lympho-histiocytic villitis and hypervascular villihttps://obstetricalpathology.com/wp-content/uploads/2012/12/cs12-7624_10xplac.jpgFigure 2Placental villi from an aborted Potter syndrome fetus at 23 weeks gestationhttps://obstetricalpathology.com/wp-content/uploads/2012/12/cs12-7608_10xpla2.jpgFigure 1Dilated villous capillaries above a retroplacental hematoma2015-11-30T18:06:25+00:00monthlyhttps://obstetricalpathology.com/2012/12/03/maternal-floor-infarction-mfi-demystified/2012-12-03T13:24:58+00:00monthlyhttps://obstetricalpathology.com/2012/11/30/machin-comment-on-stillborn-case-posted-on-new-venue-post-see-there/2012-11-30T13:23:51+00:00monthlyhttps://obstetricalpathology.com/2012/11/26/is-preterm-labor-evolutionarily-beneficial/2012-11-26T17:55:06+00:00monthlyhttps://obstetricalpathology.com/2012/11/20/stillbirth-case-umbilical-cord-accident/https://obstetricalpathology.com/wp-content/uploads/2012/11/10752.jpg10752Fig 2: mural thrombus in a surface vesselhttps://obstetricalpathology.com/wp-content/uploads/2012/11/10755.jpgfig 1Avascular villi on the right, slightly hypervascular villi on the left2012-11-20T20:12:18+00:00monthlyhttps://obstetricalpathology.com/2012/11/14/limits-in-the-microscopic-understanding-of-chorioamnionitis/2012-11-14T15:17:16+00:00monthlyhttps://obstetricalpathology.com/2012/11/14/long-cord-short-cord/2012-11-14T15:13:54+00:00monthlyhttps://obstetricalpathology.com/2012/11/14/historyand-future-of-perinatal-asphyxia-research/2012-11-14T15:13:00+00:00monthlyhttps://obstetricalpathology.com/2012/11/12/new-venue/2012-11-29T19:18:03+00:00monthlyhttps://obstetricalpathology.comdaily1.02024-12-11T18:19:27+00:00